References
- 1 Bonazzi M, Cossart P. Impenetrable barriers or entry portals? The role of cell–cell adhesion during infection. J. Cell Biol.195(3),349–358 (2011).
- 2 Lütschg V, Boucke K, Hemmi S, Greber UF. Chemotactic antiviral cytokines promote infectious apical entry of human adenovirus into polarized epithelial cells. Nat. Commun.2,391 (2011).
- 3 Ludlow M, Rennick LJ, Sarlang S et al. Wild-type measles virus infection of primary epithelial cells occurs via the basolateral surface without syncytium formation or release of infectious virus. J. Gen. Virol.91(Pt 4),971–979 (2010).
- 4 Shirogane Y, Takeda M, Tahara M, Ikegame S, Nakamura T, Yanagi Y. Epithelial–mesenchymal transition abolishes the susceptibility of polarized epithelial cell lines to measles virus. J. Biol. Chem.285(27),20882–20890 (2010).
- 5 Muhlebach MD, Mateo M, Sinn PL et al. Adherens junction protein nectin-4 is the epithelial receptor for measles virus. Nature480(7378),530–533 (2011).
- 6 Noyce RS, Bondre DG, Ha MN et al. Tumor cell marker PVRL4 (nectin 4) is an epithelial cell receptor for measles virus. PLoS Pathog.7(8),E1002240 (2011).
- 7 Antar AA, Konopka JL, Campbell JA et al. Junctional adhesion molecule-A is required for hematogenous dissemination of reovirus. Cell Host Microbe5(1),59–71 (2009).
- 8 Reuter D, Schneider-Schaulies J. Measles virus infection of the CNS: human disease, animal models, and approaches to therapy. Med. Microbiol. Immunol.199(3),261–271 (2010).
- 9 Rima BK, Duprex WP. New concepts in measles virus replication: getting in and out in vivo and modulating the host cell environment. Virus Res.162(1–2),47–62 (2011).
- 10 Li K, Li NL, Wei D, Pfeffer SR, Fan M, Pfeffer LM. Activation of chemokine and inflammatory cytokine response in hepatitis C virus-infected hepatocytes depends on Toll-like receptor 3 sensing of hepatitis C virus double-stranded RNA intermediates. Hepatology doi:10.1002/hep.24763 (2012) (Epub ahead of print).
- 11 Marukian S, Andrus L, Sheahan TP et al. Hepatitis C virus induces interferon-lambda and interferon-stimulated genes in primary liver cultures. Hepatology54(6),1913–1923 (2011).